Angioedema, use of ACE inhibitors, and SARS-CoV-2 infection
DOI:
https://doi.org/10.32818/reccmi.a6n1a4Keywords:
angioedema, ACE inhibitors, bradykinin, SARS-CoV-2 infection.Abstract
The SARS-CoV-2 infection, as well as other viral infections, can lead to a considerable variety of cutaneous manifestations. However, angioedema is not usual, generally showing up associated with urticaria and respiratory symptoms. Its emergence in patients who are under ACE inhibitors is not common. It seems that the mechanism by which the virus and ACE inhibitors produce angioedema is similar, both causing dysregulation of ACE-2 receptors and increasing bradykinin levels. It is reasonable to believe that SARS-CoV-2 may act as a potential trigger when combined with the use of these products.
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Copyright (c) 2021 Tamara Gómez-Traveso, Raquel Jaso-Tejera, Ana González-Torre-González, Elena Casuso-Sáenz, Nuria Díez-Herrán
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